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Additionally, hypoxia-inducible factor 1-alpha (HIF-1)-mediated hypoxia induced upregulation of BNIP3 and NIX/BNIP3L, which activated cell death pathways in solid tumors (Sowter et al., 2001)

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Cigarette smoke disrupts gutlung axis communication through microbial metabolite alterations driving pulmonary inflammation and cancer progression Interestingly, accumulating investigations demonstrate that cigarette smoke distorts gutlung axis communication by altering microbial metabolite profiles, creating a pro-inflammatory milieu that fuels pulmonary deterioration in COPD and promotes oncogenic niches in lung adenocarcinoma (LUAD)