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ALCAR also blocked the activation of NF-B and ICAM-1 and inhibited inflammatory angiogenesis in vivo , interfering with endothelial cell and macrophage recruitment [30]

Insights on persistent airway infection by non-typeable Haemophilus influenzae in chronic obstructive pulmonary disease

For example, when activation of endothelial TLR2 induces production of inflammatory mediators and chemoattractants, such as IL-6, granulocyte colony stimulating factor (G-CSF), and IL-8 (Wilhelmsen et al., 2012), which can further immune responses by acting on neighboring microglia and perivascular macrophages

Abe K, Maeda Y, Matsuzaki C, et al

CD is an inflammatory bowel disease that exerts gastrointestinal tract and systemic effects