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(Science-Based Medicine) Potential thyroid, kidney or metabolic disturbances some anecdotal reports and reviews raise these as possible but under-studied risks

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R Cytosolic GSTs function as homodimers or heterodimers with a conserved N-terminal domain containing the GSH binding site (G-site) and a variable C-terminal domain containing the hydrophobic substrate binding site (H-site)

Homocysteine aggravates cortical neural cell injury through neuronal autophagy overactivation following rat cerebral ischemia-reperfusion

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