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Even aggressive interventions may provide only temporary relief because the pain-processing circuits themselves remain dysregulated

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BPC-157 (as PL-14736) was evaluated in early human inflammatory-bowel-disease/ulcerative-colitis trials and reportedly in multiple-sclerosis trials

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Preclinical research suggests it may influence nitric oxide pathways, vascular endothelial growth factor, and cellular survival mechanisms involved in wound healing and tissue regeneration