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The concern isn't BDNF elevation itself but the possibility of compensatory downregulation of endogenous BDNF production if exogenous signaling is chronically sustained

Thoudam T, Chanda D, Lee J, Jung M, Sinam I, Kim B, Park B, Kwon W, Kim H, Kim M (2023) Enhanced Ca2+-channeling complex formation at the ER-mitochondria interface underlies the pathogenesis of alcohol-associated liver disease

(PubMed) Pearce EN, Leung AM, Blount BC, et al