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As oxidative stress increases, further stimulation of pro-inflammatory cytokines such as TNF- and IL-1 increases and contributes to synaptic dysfunction and neuronal degeneration ( These studies suggest that the loss of antioxidant defense is an early biochemical event rather than a late-stage consequence of neuronal death

However, no non-synonymous SNPs (single-nucleotide polymorphism), i.e

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Resveratrol and EGCG also interfere with this axis by reducing Th17 cell differentiation and IL-17 signaling [103]