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In common with a number of other conditions, for example, Sjogren disease (1), one prominent clinical manifestation is a dysfunction of small nerve fibers that occurs in a patchy, nonlength-dependent manner (small fiber neuropathy [SFN])

Mechanistically, they demonstrated that STUB1 promoted polyubiquitination of GPX4 on lysine 191, leading to its degradation by the ubiquitin proteasome pathway

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The application of this coenzyme recycling strategy for the enzymatic detoxification of mycotoxins is worth exploring

This is important because GLP-1 drugs were originally designed to have a long half-life in blood to maximise their efficacy in the treatment of diabetes which prevents them from entering the brain (Hlscher)