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Overall, these results indicated that RhoA regulated ferroptosis by affecting mitochondrial function and oxidative stress

Cp1D could also amidate the terminal carboxylate of l-Asp-l-Phe, l-Asp-l-Leu and glutaryl-l-Leu with moderate activity to form isopentyl amidated dipeptides (Supplementary Fig

Ferroptosis triggers can be divided into those that act through the cystine/glutamate antiporter system, such as erastin, and those that act by inhibiting the activity of glutathione peroxidase, such as Ras Selective Lethal 3 [73, 74]

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[PubMed: 15054433] 102