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It hyperactivates dehydrogenases in the tricarboxylic acid (TCA) cycle, drives excessive electron transport and ROS generation, induces sustained opening of the mitochondrial permeability transition pore (mPTP), and activates Ca 2+ -dependent proteases and nucleases that disrupt cellular architecture, ultimately leading to intrinsic apoptosis ( Importantly, this Ca 2+ dysregulation also extends beyond the mitochondria and affects tissue-level physiology

Angiogenic peptides (BPC-157), monitor retinal perfusion if technology available

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