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In a mouse model with I/R injury, the combination of cyclosporine A (CsA) and DFX synergistically inhibits I/R-induced iron overload, lipid peroxidation, and cardiomyocyte ferroptosis, reducing infarct size and improving poor cardiac remodeling after I/R injury

Engelsberger V, Gerhard M, Mejias-Luque R

Furthermore, we confirm that the initial glutathionylation of the glutaredoxin moiety and not the glutathione transfer to the roGFP2 moiety is rate-limiting for the oxidation of fusion constructs at physiological or low micromolar GSSG concentrations, thus allowing glutaredoxin structure-function analysis in vivo

10.1016/j.foodres.2024.114508 219 ZhaoxiaW.SiyaoS.JiaqianJ.TanD.DonghuaJ.BingB.et al (2014)

Metabolic effects of a growth hormonereleasing factor in patients with HIV