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Helicobacter pylori-mediated oxidative stress and gastric diseases: A review

In SLE, mitochondrial dysfunction contributes to oxidative stress, altered apoptosis, mtDNA mutations, and impaired immune complex clearance, aggravating the loss of self-tolerance

Yang D, Liu H, Cai Y, Lu K, Zhong X, Xing S, Song W, Zhang Y, Ye L, Zhu X, et al

**The result depends on individual metabolism

100,101,102 A similar biphasic pattern occurs in MASLD: mitophagy is enhanced during early disease to counteract lipid excess and maintain mitochondrial homeostasis, but prolonged metabolic stress suppresses this adaptive response