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*Correspondence: Worachart Lert-Itthiporn, [email protected] Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

Collaborative initiatives should be promoted to bridge research gaps and expedite the development of novel therapies for OA

This mechanism establishes OLA as a promising anti-fibrotic therapeutic candidate ( Mitochondria-targeted antioxidants or CYP2E1 inhibition prevents this toxicity, highlighting CcO dysfunction as a key pathological mechanism ( Daphnia magna through its reactive metabolite NAPQI

Poor responders or those with particularly stubborn chronic conditions may benefit from longer treatment phases before breaks

This provides evidence for the positive effect of glutathione on VD metabolism and vitamin D receptor gene expression