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Experimental observations suggest that it participates in coordinated cellular repair processes by influencing fibroblast activity, modulating angiogenic signaling, and regulating the expression of structural extracellular matrix components such as collagen and elastin

In published literature the individual components appear in models of connective-tissue repair, angiogenesis, cell migration, and copper-dependent extracellular-matrix signaling

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Lower cortisol concentrations are associated with higher pain intensity, which is a symptom of fibromyalgia

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