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A key mechanism involves the transforming growth factor-beta (TGF-) signaling pathway

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However, while the effect of IR on GPX4 activity may vary by cell type, it is well established that increased GSH and GPX4 levels are protective against IR-induced ferroptosis in both healthy and malignant cells [35, 51, 56, 57]

Cortistatin promotes this synchronization directly Homeostatic regulation -- preprocortistatin mRNA is upregulated after sleep deprivation and follows a circadian rhythm, indicating that cortistatin functions as an endogenous homeostatic sleep factor BDNF-dependent expression -- brain-derived neurotrophic factor regulates cortistatin expression in an activity-dependent manner, linking neuronal activity during wakefulness to subsequent sleep drive Cortistatin's receptor pharmacology is well-defined