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the D-Nle Trp- Ile-NH-(CH 2 )5-CONH 2 analog which exhibits intermediate inhibition of dimerization, predictably has a moderate affinity for HGF and a moderate ability to inhibit HGF-dependent processes ( Figures 25, 26, 27)

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At present, we still have a very limited knowledge of the transport processes for UA into and within the human brain