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With the disruption of dopaminergic signaling as the main culprit of motor symptoms within PD, pharmacological interventions primarily aim to restore dopamine levels, enhance its action, or modulate related pathways to alleviate motor symptoms

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Molecular mechanism of selenoprotein P synthesis

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Inactivation of DHODH in cancer cells with low GPX4 expression significantly increased intracellular lipid peroxidation, while in cancer cells with high GPX4 expression, DHODH inactivation synergized with inducers to enhance ferroptosis